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Forscher verjüngen menschliche Zellen um mehr als 60 Jahre
Forscher der Universität und des Universitätsklinikums Bonn haben unreife rote Blutzellen eines 80-Jährigen in neurale Stammzellen mit einer molekularen Alterssignatur von unter 20 Jahren umprogrammiert. Das schrittweise Verfahren soll die Erforschung des Alterns und altersbedingter Krankheiten wie Alzheimer erleichtern.
Ist Schwesterlichkeit das Geheimnis eines langen Lebens? Das sardische Dorf Bolotana als mögliche blaue Zone für langlebige Frauen
Protein TIMP2 verjüngt Immunfunktionen alternder Gehirnzellen bei Mäusen
Neues Medikament kehrt schwere Fettlebererkrankung durch Darmreparatur um
Gehirne mancher Erwachsenen wirken älter als ihr tatsächliches Alter, und die Darmbiologie könnte eine Rolle spielen
Zelluläres Recycling verändert sich im Alter anders als angenommen
Wissenschaftler entdecken verborgenen Alterstreiber und Aminosäure gegen Gedächtnisverlust bei Mäusen
US-Behörde plant Ausweitung von Ausnahmen für giftige Chemikalien in Lebensmitteln
GLP-1-Abnehmmittel: Forscher entdecken Wirkung auf biologisches Alter und weitere Nebenwirkungen
Riesenschildkröte Jonathan ist mit 194 Jahren genetisch aktiv wie ein Jungtier
Sardisches Dorf könnte neue «Blue Zone» für Hundertjährige sein
Natürliche Transfette aus Milchprodukten erhöhen laut großer Analyse nicht das Krankheitsrisiko
Forschungs-Feed
Direkt aus den Feeds von Journalen und Instituten, in Originalsprache. Die Relevanz kommt aus einem Stichwortfilter.
Zweifelhafter Trend: »Dopaminfasten ist völliger Unsinn«
Der Neurobiologe Martin Korte hält nichts von Dopaminfasten. Was dem Gehirn stattdessen hilft, Gewohnheiten zu verändern und effektiver zu arbeiten.
L-Nutra Health launches metabolic programme to UK clinics
L-Nutra Health has launched a clinician-led metabolic health programme in the United Kingdom, making it available to clinics and medical practices for the first time. The programme is built on more than 25 years of research led by Dr Valter Longo and centres on the Fasting Mimicking Diet, which the company claims is a patented, […] The post L-Nutra Health launches metabolic programme to UK clinics appeared first on Longevity.Technology.
Reprogramming the gut–brain axis in neurodegeneration: comparative microbiome, metabolite, immunometabolic, polyphenol, probiotic, and epigenetic mechanisms in Alzheimer’s and Parkinson’s diseases
The gut–brain axis has emerged as a central mechanistic framework linking dietary bioactives, microbial ecology, and neurodegenerative disease progression. Similar microbiome-related mechanisms have also been implicated in colorectal cancer, suggesting potential shared links between microbial dysbiosis, inflammation, and disease progression. This study synthesizes preclinical evidence examining how polyphenols, polyphenol-derived compounds, and advanced delivery systems, including nanoparticles and synbiotics, modulate Alzheimer’s disease (AD) and Parkinson’s disease (PD) through microbiome-dependent mechanisms. Evidence from transgenic mice, toxin-induced rodent models, and Drosophila systems was compared across microbiome alterations, microbial metabolites, signaling pathways, epigenetic regulation, and functional outcomes. Across AD and PD models, dietary bioactives consistently promo
Microbial metabolites as modulators of gut–brain communication in neurodegeneration: from intestinal sensing to disease initiation and progression
How heterogeneous gut metabolite alterations relate to Parkinson’s disease (PD) and Alzheimer’s disease (AD) remains unresolved. We propose a framework in which intestinal sensing of indole derivatives, short-chain fatty acids and bile acids links microbial function to barrier regulation and gut–brain communication. Integration denotes tissue-level convergence of aryl hydrocarbon receptor (AhR), pregnane X receptor (PXR) and metabolite-responsive G-protein-coupled receptor responses across epithelial and neighbouring immune cells, rather than an established same-cell receptor complex. Tryptophan metabolism provides an analytical focus connecting microbial indole production with host kynurenine and serotonin pathways; metabolite concentrations alone do not establish pathway flux or receptor activity. We hypothesise that suppressed indole-associated epithelial signalling, barrier hyperperm
Millions take fish oil for brain health. A two-year study found no benefit
A surprising two-year clinical trial found that fish oil supplements successfully delivered omega-3s to the brain but failed to improve memory or protect against Alzheimer's-related brain shrinkage. Researchers suspect that getting these nutrients through a healthy diet may offer benefits that supplements alone cannot provide.
Zealand taps Generate’s AI for obesity proteins in nearly $1.2b deal
Zealand Pharma and Generate Biomedicines will design multispecific proteins for metabolic disease, in a deal with nearly $1.2b in milestones. Zealand Pharma has been a peptide company for more than 25 years, and now the Copenhagen outfit is asking an AI specialist to help it branch into proteins. The announcement came on 13 October. Zealand […] The post Zealand taps Generate’s AI for obesity proteins in nearly $1.2b deal appeared first on Longevity.Technology.
Loss of Oocyte Ribosomal Function as a Contribution to Female Reproductive Aging
Why, mechanistically, is female reproductive aging so accelerated in relation to the rest of the aging process? Researchers here point to a role for a progressive loss of ribosomes in oocytes. Cells contain many ribosomes, vital components of the cell where messenger RNA is translated into protein, and usually synthesize more as needed. Oocytes do not tend to synthesize new ribosomes, however, one of many differences in behavior intended to retain a much higher standard of cell quality during the reproductive lifespan. Female germline cells are among the most protected of all cell types, and have to be in order to maintain a sufficiently low level of reproductive failure for evolutionary success. Here, it is suggested that this ribosomal approach to increased cell quality produces […]
Natural compound could fight rheumatoid arthritis at its source
A natural compound extracted from a medicinal plant has shown surprising promise against rheumatoid arthritis. In rats, it reduced joint swelling, protected cartilage, and calmed the immune responses responsible for chronic inflammation. Scientists discovered that the compound works by targeting a specific protein and restoring balance to fatty acid metabolism, revealing a previously underexplored way to fight the disease.
Cerebrospinal fluid neurofilament heavy protein levels associated with domain-specific cognitive decline in Alzheimer’s disease
BackgroundNeurofilament proteins are fluid biomarkers of neuroaxonal damage in various neurological diseases. However, it remains to be elucidated whether CSF neurofilament heavy (NfH) protein levels correlate with cognitive decline and progression in Alzheimer’s disease (AD).MethodsThis study included 165 cognitively unimpaired (CU) participants, 385 participants with mild cognitive impairment (MCI), and 128 participants with AD. To examine correlations between CSF NfH levels and baseline cognitive measures, Pearson’s correlation was used. Linear mixed-effects models were fitted to assess the relationship between elevated CSF NfH levels and longitudinal cognitive decline in the CU, MCI and AD groups. Kaplan–Meier survival analyses and Cox proportional hazards models were used to assess the clinical progression in the CU and MCI groups separately.ResultsIn the MCI group, higher CSF NfH l
Insilico’s AI-built pill cuts colitis scores by 60% in animal tests
Insilico Medicine nominates ISM9546, an oral VAV1 degrader designed with generative AI, as a preclinical candidate for autoimmune disease. Some proteins have a reputation for being impossible to hit with a drug. VAV1 is one of them, and Insilico Medicine says an AI-designed pill has now taken a decent swing at it. The Hong Kong-listed […] The post Insilico’s AI-built pill cuts colitis scores by 60% in animal tests appeared first on Longevity.Technology.
Scientists find 10 different diets linked to slower aging
A study by DZNE finds that healthy eating is associated with slower biological aging—and that there is more than one way to eat healthily. The researchers examined ten dietary patterns considered healthy, including the Mediterranean, Nordic, and a plant-based diet, as well as the DASH diet, which is designed to help lower blood pressure. The findings are published in the journal Nature Communications.
Circulating inflammatory biomarkers as adjusted prognostic predictors of post-stroke cognitive impairment: a comparative meta-analysis of cytokines and lymphocyte-based ratio indices in prospective cohorts
IntroductionSystemic inflammation is implicated in post-stroke cognitive impairment (PSCI). Two families of blood markers have been studied as predictors: cytokines (interleukin-6, C-reactive protein) and lymphocyte-based ratio indices from the full blood count, including the neutrophil-to-lymphocyte ratio, the systemic immune-inflammation index (SII) and the globulin-to-lymphocyte ratio (GLR). Existing syntheses pool unadjusted marker differences within a single family, so their relative adjusted associations with PSCI are unknown.MethodsWe systematically reviewed strictly prospective cohorts reporting a covariate-adjusted association between a circulating inflammatory marker and PSCI. Estimates were harmonised to the odds ratio per one standard deviation (SD) increase, deriving SDs where needed from medians and interquartile ranges and rescaling category contrasts by category-spacing f
Small-fiber degeneration, sensory dysfunction, and nociception comorbidity in central neurodegenerative disease
Progressive neurodegenerative disease typically calls for clinical effort to improve cognitive and motor deficits. It is also focused on improving patients' quality of life. However, across these dementias and other diseases, the underlying causes and potential mechanisms remain unclear. Most experimental and clinical research focuses on the central nervous system, examining pathogenic proteins such as tau and amyloid beta, as well as the potential cascade following mitochondrial damage and neuroinflammation. Although peripheral involvement, including gut microbiome influences and other visceral organs, is well-documented, the peripheral nervous system remains an understudied component of these progressive diseases. As peripheral terminals of primary afferent neurons are exposed to inflammatory or toxic mediators, these neurons respond to these threats via neuron-specific phenotypic chan
Creation of Chimeric Antigen Receptor Natural Killer Cells in Situ via Messenger RNA Therapy
Messenger RNA (mRNA) can be delivered into tissues and cells by encapsulating it into lipid nanoparticles (LNPs), and then injecting the nanoparticles. A target cell engulfs a LNP, and the cargo of mRNA is released into the cytoplasm where it is translated into protein. Typically a protein is manufactured in cells exposed to a LNP-mRNA therapy for a few days only before the last of the delivered mRNA is broken down and recycled, so the end result might be considered a half-way point between the characteristics of a gene therapy and the characteristics of a small molecule drug: a treatment that produces a protein of interest to cause specific desired outcomes in cell behavior, but that only lasts a little while. The lipid nanoparticle industry […]
Parkinson’s test-and-drug duo wins CPHI award
German biotech MODAG won for pairing a CE-marked diagnostic with a drug candidate that targets the same protein. The jury at CPHI Milan had a pattern to break. For three years running, the API Development and Innovation award had gone to manufacturers with new production processes. On 6 October, it went to a small German […] The post Parkinson’s test-and-drug duo wins CPHI award appeared first on Longevity.Technology.
Scientists find a way to fight Alzheimer’s brain damage without removing toxic tau
Scientists have found a promising way to protect the brain from Alzheimer’s-related damage by blocking immune cells that contribute to the destruction of neurons. In mice, the treatment preserved roughly 40% more tissue in memory centers and improved memory, even without removing harmful tau protein buildup.
An RNA Regulator Mitigates Senescence in Blood Vessels
Researchers publishing in Aging Cell have found that the regulatory protein MTR4 fights DNA damage in the blood vessel lining but decreases with aging. Vulnerable by […]
VectorY triples ALS trial before efficacy data
VectorY is growing PIONEER-ALS from 12 to 36 participants to test VTx-002, an antibody therapy aimed at the protein TDP-43. VectorY Therapeutics has just tripled the size of its ALS trial, and it did so before seeing any efficacy data. The Phase 1/2 PIONEER-ALS study of VTx-002 will now enroll 36 people instead of 12 […] The post VectorY triples ALS trial before efficacy data appeared first on Longevity.Technology.
Why some people escape Alzheimer’s despite carrying a high-risk gene
Scientists have discovered why some people with a high genetic risk of Alzheimer’s remain mentally sharp into their 80s, uncovering a rare mutation that protects the brain from harmful protein buildup. The findings reveal a potential new way to prevent early brain damage and could pave the way for treatments that mimic this natural defense.
Just 3,000 steps a day may help slow Alzheimer's, scientists find
Walking just 3,000 steps a day could help slow Alzheimer’s-related brain changes, according to new research. Scientists discovered that physical activity was linked to less buildup of harmful tau proteins and nearly half the cognitive and functional decline, even in people already showing early signs of the disease. The strongest benefits appeared at 5,000 to 7,500 daily steps, suggesting that the popular 10,000-step goal may not be necessary.
Physical exercise: a multi-target strategy for the prevention and treatment of Alzheimer’s disease
Alzheimer’s disease (AD) is a devastating age-related neurodegenerative disease characterized by irreversible cognitive dysfunction and neuronal loss. The complex pathogenesis of AD involves core proteinopathies of amyloid-β (Aβ) and tau, chronic neuroinflammation, and extensive cerebral metabolic disorders, which collectively drive progressive neuronal damage and disease deterioration. Currently, effective disease-modifying drugs for AD are still lacking, and physical exercise has been recognized as a safe and effective non-pharmacological intervention to prevent and alleviate AD pathological progression. Specifically, exercise inhibits abnormal Aβ deposition and tau hyperphosphorylation, suppresses microglial overactivation and sustained neuroinflammatory responses, and remodels cerebral metabolic homeostasis by improving glucose utilization, balancing lactate metabolism, restoring mit
Brain cells meant to protect us may be fueling cognitive decline
Scientists have uncovered a surprising cause of age-related cognitive decline involving brain cells previously thought to be entirely beneficial. As these cells become dysfunctional, they can produce abnormal amounts of myelin, disrupting nerve fibers and potentially contributing to worsening memory and thinking skills.
Scientists discover how exercise reverses muscle aging
Scientists have uncovered a hidden biological mechanism that helps explain how exercise can reverse some of the damage caused by muscle aging. The key is a gene called DEAF1, which becomes overactive with age and interferes with the muscles’ ability to clear damaged proteins and repair themselves. Exercise can help restore this balance, allowing older muscles to rebuild and regain strength, although the benefits may diminish when the system becomes too disrupted.
Blood clues to why Alzheimer’s drug results vary
A Lancet Neurology study of 130 blood proteins in patients receiving lecanemab suggests amyloid clearance and cognitive response differ. Anti-amyloid drugs have moved into routine care for some people with early Alzheimer’s, but the benefit isn’t the same for everyone who takes them. A new study in The Lancet Neurology, built around a blood-testing platform […] The post Blood clues to why Alzheimer’s drug results vary appeared first on Longevity.Technology.
Menopausal Serum Protein Changes May Predict Brain Health
A recent analysis of serum protein changes identified 16 proteins related to inflammatory, synaptic, metabolic, and Alzheimer’s disease processes whose levels change during the menopausal transition [1]. These changes were linked to cognitive health later […]
Arvinas takes its Parkinson’s degrader to Seoul
Arvinas presented early biomarker data on ARV-102, an oral drug built to clear the LRRK2 protein, at a major Parkinson’s meeting. Five minutes. That’s the slot Arvinas was given to present late-breaking data from its Phase 1 trial of ARV-102 in people with Parkinson’s disease. The stage was the 2026 International Congress of Parkinson’s Disease […] The post Arvinas takes its Parkinson’s degrader to Seoul appeared first on Longevity.Technology.
Scientists may have overlooked a hidden Alzheimer's culprit for decades
For decades, scientists have focused on amyloid beta as a major culprit behind Alzheimer’s disease, spending billions of dollars developing treatments that have often delivered disappointing results. Now, researchers at UC Santa Cruz are drawing attention to a largely overlooked peptide called P3 that could also play a role in the disease. Previously believed to be harmless, P3 can form harmful-looking protein clumps even faster than amyloid beta and may also damage brain cells.
Pathological remodeling and functional evolution of the corneal nervous system in ocular surface dyshomeostasis
The corneal nerve plexus is essential to ocular surface homeostasis through sensory detection, trophic support, epithelial repair, blinking, and lacrimal reflexes. In dry eye disease (DED), chronic hyperosmolar, inflammatory, mechanical, and age-related stressors alter nerve morphology and function, contributing to reduced nerve density, aberrant regeneration, microneuroma formation, sensory loss, and peripheral or central sensitization. This review summarizes the ultrastructure of corneal innervation and the molecular basis of corneal sensory transduction, with emphasis on TRPM8, TRPV1, TRPA1, Piezo2, and acid-sensing ion channels. It then examines bidirectional neuroimmune signaling involving dendritic cells, cytokines, neuropeptides, and diminished neurotrophic support. We further discuss the relationship between age-related corneal hypoesthesia, local small-fiber degeneration, and sy
Low-Fat Vegan Diet Shows Benefits in Type 2 Diabetes
A human trial found that even without caloric restriction, a low-fat, plant-based diet improved blood sugar control, reduced liver fat, and lowered the quantity of advanced glycation end products (AGEs) in the skin [1]. An all-you-can-eat vegan buffet Type 2 […]
Scientists find hidden driver of aging and a simple amino acid that fights memory loss in mice
Scientists discovered that declining levels of a protein called Menin may help drive aging, and restoring it in older mice reversed several age-related changes in just 30 days. Even more surprisingly, supplementing with a simple amino acid called D-serine improved memory and learning, offering a potential new approach to age-related cognitive decline.
Arguing that Methionine Restriction is Actually Cysteine Restriction
A reduced intake of calories triggers protective responses to low nutrient availability, dramatically altering the operation of cellular metabolism. For so long as adequate levels micronutrients are still ingested, this response to calorie restriction is beneficial. The cell maintenance processes of autophagy operate more efficiently, for example, and this improved autophagy is thought to be the primary mechanism by which calorie restriction improves health and slows aging. This response to calorie restriction is triggered by a variety of independent sensors that assess the availability of various components of the diet, where essential amino acids are one of the more important components in this respect. Researchers have assessed the metabolic response to diets low in the essential amino acid methionine, and found it to produce broadly […]
A SynNotch-PiggyBac gene circuit for inflammation-responsive IL-10 delivery in Alzheimer’s disease: a hypothesis and design framework
Chronic neuroinflammation driven by sustained microglial and astrocyte activation contributes to Alzheimer’s disease progression, yet no therapeutic strategy addresses it with precision or durability in the brain. Interleukin-10 occupies an unresolved position in this problem. Constitutive interleukin-10 overexpression in amyloid precursor protein transgenic mice increased plaque burden by suppressing microglial phagocytic activity, while interleukin-10 deletion has been reported to restrict amyloidosis in one setting and to worsen tau hyperphosphorylation in another. We interpret this pattern as evidence that interleukin-10 carries opposite signs on the two principal pathologies of Alzheimer’s disease, so that any therapy holding it at a fixed concentration is committed in advance to being wrong about one of them. We therefore propose a falsifiable hypothesis: conditional, inflammation-
Tazbentetol protects eye cells in diabetic preclinical study
A peer-reviewed preclinical study published in Experimental Eye Research evaluated daily Tazbentetol in diabetic db/db mice over eight weeks and, according to the company, found preservation of retinal ganglion cell function and survival, restoration of synaptic protein expression, reduced reactive gliosis and cell death markers, improved optic nerve myelination, reversal of decreases in pattern electroretinogram […] The post Tazbentetol protects eye cells in diabetic preclinical study appeared first on Longevity.Technology.
Reduced serum LEFTY2 and disease-dependent biomarker associations in Alzheimer’s disease: an exploratory study
Reliable blood-based biomarkers are increasingly needed for the detection and monitoring of Alzheimer’s disease (AD). Left–right determination factor 2 (LEFTY2) is a secreted protein of the transforming growth factor-β (TGF-β) superfamily. Evidence from AD-related neuronal models suggests that LEFTY2 promotes neuronal survival, attenuates amyloid- and tau-related pathology, and suppresses inflammatory responses, highlighting its potential relevance to AD. However, its clinical significance and relationship with AD-related blood biomarkers remain unclear. In this study, we investigated the association between serum LEFTY2 and and biomarkers associated with amyloid pathology, tau pathology, neuroinflammation, and neurodegeneration in patients with AD and age-matched cognitively normal (CN) controls. Biomarker levels were quantified using enzyme-linked immunosorbent assays. Serum LEFTY2 lev
An Aging Clock Based on Images of Hematopoietic Stem Cell Nuclei
Any sufficiently complex biological data that changes with age can be used to construct an aging clock. Here, researchers report on a clock derived from images of the nuclei of hematopoietic stem cells. Nuclear DNA exhibits structural changes in response to circumstances, regulated by epigenetic decorations to DNA and its supporting proteins, as regions unfold or are compacted. The structure of DNA determines which gene sequences are exposed to translation machinery, and thus whether or not a given gene is expressed. Some of these structural changes are characteristic of aging, and thus machine learning approaches should be able to visualize age-related differences in imaging. The functional decline of the hematopoietic system during aging affects organismal function and contributes to reduced healthspan. Quantifying hematopoietic aging holds […]
Your gut may be making a molecule that raises Alzheimer’s risk
Scientists have linked a gut bacterial compound called ImP to greater Alzheimer’s risk, harmful brain protein buildup, and faster cognitive decline. The finding raises the possibility that reducing ImP in the blood could one day help slow or prevent dementia.
MitoRx presents preclinical data showing restored liver mitochondria
MitoRx Therapeutics presented preclinical transmission electron microscopy data for its lead small molecule, MTRX31, at the 62nd EASD Annual Meeting in Milan, with an oral presentation on 30 September 2026. In a diet-induced obesity mouse study carried out at the University of Oxford, the company reports that MTRX31 restored mitochondrial density per unit tissue area […] The post MitoRx presents preclinical data showing restored liver mitochondria appeared first on Longevity.Technology.
Dexmedetomidine as a potential neuroprotective agent against Alzheimer’s disease: molecular mechanisms and therapeutic perspectives
Alzheimer’s disease (AD) is a neurodegenerative disorder characterized by extracellular deposition of amyloid-β (Aβ), intracellular hyperphosphorylation of microtubule-associated protein tau and chronic neuroinflammation. At present, effective disease-modifying therapies for AD remain limited. Dexmedetomidine (Dex), a highly selective α2-adrenergic receptor agonist, has been reported in recent years to exert significant neuroprotective effects. Accumulating evidence indicates that Dex may ameliorate AD pathology through multiple mechanisms. In terms of Aβ metabolism, Dex reduces Aβ production by downregulating β-site amyloid precursor protein-cleaving enzyme 1 (BACE1) expression. With respect to tau pathology, Dex may counteract abnormal tau hyperphosphorylation in AD-related models, although relatively high experimental doses have also been associated with increased tau phosphorylation.
Associations between inflammatory-lipid indices and clinical outcomes in ischemic stroke patients treated with mechanical thrombectomy
PurposeInflammation and dyslipidemia are critical in post-stroke prognosis. We conducted a comprehensive analysis of six common inflammatory-lipid indices to evaluate the associations between these indices and four clinical outcomes after mechanical thrombectomy (MT) for acute ischemic stroke (AIS), and to identify generalizable consistently associated markers for all four clinical outcomes.MethodsIn this retrospective studyassociations between inflammatory-lipid indices, namely monocyte-to-high-density lipoprotein cholesterol ratio (MHR), platelet-to-HDL-C ratio (PHR), neutrophil-to-HDL-C (NHR), lymphocyte-to-HDL-C ratio (LHR), fibrinogen-to-HDL-C ratio (FHR), and C-reactive protein-to-HDL-C ratio (CRP/HDL-C), and four post-MT clinical outcomes comprised hemorrhagic transformation (HT), malignant cerebral edema (MCE) 90-day unfavorable outcome and all-cause mortality were evaluated usin
Hydrogen gas and olfactory dysfunction in Alzheimer’s disease: the Hydrogen–Olfactory–Limbic hypothesis
Olfactory dysfunction is increasingly recognized as an early clinical feature of Alzheimer’s disease (AD), often preceding measurable cognitive impairment and associated with subsequent cognitive decline. Despite this relatively consistent clinical association, the anatomical, cellular, and physiological substrates underlying olfactory dysfunction in AD remain poorly understood. Post-mortem human studies have identified amyloid-β (Aβ) and tau pathology in olfactory structures, including the olfactory epithelium and anterior olfactory nucleus. However, the available anatomical evidence remains limited, and the presence of these pathological proteins does not establish that they cause olfactory dysfunction. Likewise, it remains uncertain whether early olfactory impairment reflects peripheral sensory dysfunction, neuronal loss, synaptic or circuit abnormalities, altered higher-order olfacto
Quellen: Lifespan Research Institute · Fight Aging! · Longevity.Technology · ScienceDaily – Healthy Aging · ScienceDaily – Nutrition · ScienceDaily – Fitness · Nature – Ageing · Frontiers in Aging Neuroscience · Medical Xpress – longevity · The Guardian – Health · Spektrum der Wissenschaft · Deutsches Ärzteblatt · Quarks · Index – Tudomány